Personal profile
Education
Courses
Physiology Laboratory
Neurobiology
Keywords
- Human Physiology
- Molecular Neurobiology
- Neurobiology
Expertise related to UN Sustainable Development Goals
In 2015, UN member states agreed to 17 global Sustainable Development Goals (SDGs) to end poverty, protect the planet and ensure prosperity for all. This person’s work contributes towards the following SDG(s):
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SDG 3 Good Health and Well-being
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Collaborations and top research areas from the last five years
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Investigating Molecular Pathogenic Mechanisms Underlying Mutant Ppp2r5d-Induced Parkinson'S Disease
Wang, H.-L. (PI) & Weng, Y. H. (CoPI)
01/08/25 → 31/07/26
Project: National Science and Technology Council › National Science and Technology Council Academic Grants
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Investigating Molecular Pathogenic Mechanisms Underlying Mutant Ppp2r5d-Induced Parkinson'S Disease
Wang, H.-L. (PI) & Weng, Y.-H. (CoPI)
01/08/24 → 31/07/25
Project: National Science and Technology Council › National Science and Technology Council Academic Grants
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Investigating Molecular Pathogenic Mechanisms Underlying Genetic Mutation of Lrp10-Induced Autosomal Dominant Parkinson'S Disease.
Wang, H.-L. (PI), Chen, Y. L. (CoPI) & Weng, Y.-H. (CoPI)
01/08/23 → 31/07/24
Project: National Science and Technology Council › National Science and Technology Council Academic Grants
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Investigating Molecular Pathogenic Mechanisms Underlying Genetic Mutation of Lrp10-Induced Autosomal Dominant Parkinson'S Disease.
Wang, H.-L. (PI), Chen, Y. L. (CoPI) & Weng, Y.-H. (CoPI)
01/08/22 → 31/07/23
Project: National Science and Technology Council › National Science and Technology Council Academic Grants
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Investigating Molecular Pathogenic Mechanisms Underlying Genetic Mutation of Lrp10-Induced Autosomal Dominant Parkinson'S Disease.
Wang, H.-L. (PI), Chen, Y. L. (CoPI) & Weng, Y.-H. (CoPI)
01/08/21 → 31/07/22
Project: National Science and Technology Council › National Science and Technology Council Academic Grants
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Absence of Rab39b-induced macroautophagy impairment increases neurotoxic α-synuclein and causes degeneration of substantia nigra dopaminergic neurons in mouse model of X-linked Parkinson's disease
Chiu, C. C., Weng, Y. H., Yeh, T. H., Chen, W. S., Liu, S. Y., Chiu, T. J., Li, A. H. L. & Wang, H. L., 15 09 2025, In: Life Sciences. 377, 123789.Research output: Contribution to journal › Journal Article › peer-review
1 Scopus citations -
Downregulation of Protease Cathepsin D and Upregulation of Pathologic α-Synuclein Mediate Paucity of DNAJC6-Induced Degeneration of Dopaminergic Neurons
Wang, H.-L., Weng, Y.-H., Chiu, C.-C., Chen, Y.-L., Li, H.-L., Liu, S.-Y. & Yeh, T.-H., 18 06 2024, In: International Journal of Molecular Sciences. 25, 12, p. 1 - 23 6711.Research output: Contribution to journal › Journal Article › peer-review
Open Access4 Scopus citations -
Deficiency of RAB39B Activates ER Stress-Induced Pro-apoptotic Pathway and Causes Mitochondrial Dysfunction and Oxidative Stress in Dopaminergic Neurons by Impairing Autophagy and Upregulating α-Synuclein
Chiu, C. C., Weng, Y. H., Yeh, T. H., Lu, J. C., Chen, W. S., Li, A. H. R., Chen, Y. L., Wei, K. C. & Wang, H. L., 05 2023, In: Molecular Neurobiology. 60, 5, p. 2706-2728 23 p.Research output: Contribution to journal › Journal Article › peer-review
11 Scopus citations -
HCH6-1, an antagonist of formyl peptide receptor-1, exerts anti-neuroinflammatory and neuroprotective effects in cellular and animal models of Parkinson's disease
Wang, H. L., Cheng, Y. C., Yeh, T. H., Liu, H. F., Weng, Y. H., Chen, R. S., Chen, Y. C., Lu, J. C., Hwang, T. L., Wei, K. C., Liu, Y. C., Wang, Y. T., Hsu, C. C., Chiu, T. J. & Chiu, C. C., 06 2023, In: Biochemical Pharmacology. 212, p. 115524 115524.Research output: Contribution to journal › Journal Article › peer-review
12 Scopus citations -
TOMM40 Genetic Variants Cause Neuroinflammation in Alzheimer’s Disease
Chen, Y. C., Chang, S. C., Lee, Y. S., Ho, W. M., Huang, Y. H., Wu, Y. Y., Chu, Y. C., Wu, K. H., Wei, L. S., Wang, H. L. & Chiu, C. C., 17 02 2023, In: International Journal of Molecular Sciences. 24, 4, 4085.Research output: Contribution to journal › Journal Article › peer-review
Open Access34 Scopus citations