Projects per year
Personal profile
Education
Courses
Physiology
Physiology Laboratory
Keywords
- Human Physiology
- Molecular Neurobiology
- Neurobiology
Expertise related to UN Sustainable Development Goals
In 2015, UN member states agreed to 17 global Sustainable Development Goals (SDGs) to end poverty, protect the planet and ensure prosperity for all. This person’s work contributes towards the following SDG(s):
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SDG 3 Good Health and Well-being
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Collaborations and top research areas from the last five years
Projects
- 64 Finished
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Investigating Molecular Pathogenic Mechanisms Underlying Mutant Ppp2r5d-Induced Parkinson'S Disease
Wang, H.-L. (PI) & Weng, Y. H. (CoPI)
01/08/25 → 31/07/26
Project: National Science and Technology Council › National Science and Technology Council Academic Grants
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Investigating Molecular Pathogenic Mechanisms Underlying Mutant Ppp2r5d-Induced Parkinson'S Disease
Wang, H.-L. (PI) & Weng, Y.-H. (CoPI)
01/08/24 → 31/07/25
Project: National Science and Technology Council › National Science and Technology Council Academic Grants
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Investigating Molecular Pathogenic Mechanisms Underlying Genetic Mutation of Lrp10-Induced Autosomal Dominant Parkinson'S Disease.
Wang, H.-L. (PI), Chen, Y. L. (CoPI) & Weng, Y.-H. (CoPI)
01/08/23 → 31/07/24
Project: National Science and Technology Council › National Science and Technology Council Academic Grants
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Investigating Molecular Pathogenic Mechanisms Underlying Genetic Mutation of Lrp10-Induced Autosomal Dominant Parkinson'S Disease.
Wang, H.-L. (PI), Chen, Y. L. (CoPI) & Weng, Y.-H. (CoPI)
01/08/22 → 31/07/23
Project: National Science and Technology Council › National Science and Technology Council Academic Grants
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Investigating Molecular Pathogenic Mechanisms Underlying Genetic Mutation of Lrp10-Induced Autosomal Dominant Parkinson'S Disease.
Wang, H.-L. (PI), Chen, Y. L. (CoPI) & Weng, Y.-H. (CoPI)
01/08/21 → 31/07/22
Project: National Science and Technology Council › National Science and Technology Council Academic Grants
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FTDP-17T Mutations Promote Formation of Phosphorylated FTDP-17T TAU Oligomers That Cause Degeneration of Dopaminergic and Hippocampal Neurons via Activating ER Stress and Mitochondrial Pro-apoptotic Cascades
Wang, H. L., Weng, Y. H., Chiu, C. C., Chen, W. S., Liu, S. Y. & Yeh, T. H., 27 04 2026, In: Neurochemical Research. 51, 3, 145.Research output: Contribution to journal › Journal Article › peer-review
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Loss-of-Function (G603R) Lrp10 Fails to Downregulate mRNA of Pathologic α-Synuclein and Causes Neurodegeneration of Substantia Nigra Dopaminergic Cells in Parkinson’s Disease Knockin Mice
Wang, H. L., Liu, S. Y., Chiu, C. C., Yeh, T. H., Chen, W. S., Chiu, T. J., Yeh, Y. C., Wu, P. R. & Weng, Y. H., 20 05 2026, In: Neurochemical Research. 51, 3, 162.Research output: Contribution to journal › Journal Article › peer-review
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Lrp10 insufficiency upregulates mRNA and protein of neurotoxic α-synuclein and causes degeneration of substantia nigra dopaminergic neurons in heterozygous or homozygous Lrp10 knockout mice
Wang, H. L., Liu, S. Y., Chiu, C. C., Yeh, T. H., Chiu, T. J., Yeh, Y. C., Wu, P. R. & Weng, Y. H., 01 07 2026, In: Neurochemistry International. 197, p. 106186 106186.Research output: Contribution to journal › Journal Article › peer-review
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PARK19 truncation mutant Dnajc6 causes lysosomal deficiency-induced upregulation of pathologic α-synuclein and neurodegeneration of substantia nigra dopaminergic cells in PARK19 knockin mice
Wang, H. L., Chen, Y. L., Chiu, T. J., Chiu, C. C., Weng, Y. H., Liu, S. Y., Li, A. H. L. & Yeh, T. H., 12 03 2026, In: npj Parkinson's Disease. 12, 1, 102.Research output: Contribution to journal › Journal Article › peer-review
Open Access -
Absence of Rab39b-induced macroautophagy impairment increases neurotoxic α-synuclein and causes degeneration of substantia nigra dopaminergic neurons in mouse model of X-linked Parkinson's disease
Chiu, C. C., Weng, Y. H., Yeh, T. H., Chen, W. S., Liu, S. Y., Chiu, T. J., Li, A. H. L. & Wang, H. L., 15 09 2025, In: Life Sciences. 377, 123789.Research output: Contribution to journal › Journal Article › peer-review
8 Scopus citations