Abstract
對六十二個臨床上無法觸摸到睪丸者，施以手術治療，發有四十個為睪丸缺失者，其中九個為睪丸併副睪及輸精管完全缺失者，兩個為輸精管末端成盲狀，二十五個為睪丸缺失併精索末端成盲狀者，此定義為消失性睪丸症候群，由此解剖上的發現可建議此種睪丸曾一度存在，而其可能在產前發育的後期消失，且兩側睪丸消失者，其遺傳表型為46XY的正常男或，可說明其睪丸必在主導男性化的某時期消失，根據文獻報告，可能是由於子宮內的血管意外。而依照消失性睪丸的解剖發即睪丸缺失併精索末端成盲狀者，可以給我們在無法觸摸睪丸的手術術中一個終止尋找睪丸的訊號。
Inguinal exploration of 62 clinically impalpable undescended testes reveales 40 (64%) instances of absent testis. Nine (22.5%) of the 40 explorations showed complete absence of the tests along with the epididymis and vas deferens, six (15%) were associated with only vas ending blindly, and the other 25 (67.5%) were associated with blind-ending vas deferens and blood vessels: the “vanishing testis syndrome.” The anatomical findings of these vanishing testes showed the presence of a vas deferens and vessels lying side by side in the inguinal canal. In Addition, two patients with bilateral vanishing testis were found to be a normal 46 xy phenotypic male. Since testicular tissue is necessary in utero to stimulate external genital development, these findings, imply that testes vanished at some point following induction of masculinization, possible as a result of an intrauterine vascular accident or testicular torsion. Also, according to the anatomical findings of vanishing testis, it gave us the signal to terminate the exploration when we found absent testis with a blind-ending vas plus vessels on one or both side in an operation for impalpable testis.
Inguinal exploration of 62 clinically impalpable undescended testes reveales 40 (64%) instances of absent testis. Nine (22.5%) of the 40 explorations showed complete absence of the tests along with the epididymis and vas deferens, six (15%) were associated with only vas ending blindly, and the other 25 (67.5%) were associated with blind-ending vas deferens and blood vessels: the “vanishing testis syndrome.” The anatomical findings of these vanishing testes showed the presence of a vas deferens and vessels lying side by side in the inguinal canal. In Addition, two patients with bilateral vanishing testis were found to be a normal 46 xy phenotypic male. Since testicular tissue is necessary in utero to stimulate external genital development, these findings, imply that testes vanished at some point following induction of masculinization, possible as a result of an intrauterine vascular accident or testicular torsion. Also, according to the anatomical findings of vanishing testis, it gave us the signal to terminate the exploration when we found absent testis with a blind-ending vas plus vessels on one or both side in an operation for impalpable testis.
| Original language | American English |
|---|---|
| Pages (from-to) | 121-124 |
| Journal | 長庚醫學 |
| Volume | 17 |
| Issue number | 2 |
| State | Published - 1994 |