Exclusive homoplasmic 11778 mutation in mitochondrial DNA of Chinese patients with Leber's hereditary optic neuropathy

May Yung Yen*, Hsin Chen Lee, An Guor Wang, Wei Ling Chang, Jorn Hon Liu, Yau Huei Wei

*Corresponding author for this work

Research output: Contribution to journalJournal Article peer-review

12 Scopus citations

Abstract

Purpose: To investigate the degree of heteroplasmy of the 11778 mtDNA mutation in Chinese patients with Leber's hereditary optic neuropathy (LHON). Methods: Seventeen Chinese Leber's pedigrees, including 24 patients, 17 unaffected maternal lineages, 4 internal controls, and 6 unrelated controls, were screened for the 11778 mtDNA mutation. This was carried out by analysis of the restriction fragment length polymorphism, single-strand conformation polymorphism, and DNA sequencing. Results: All patients and unaffected maternal lineages, regardless of their symptoms, had homoplastic 11778 mtDNA mutation, which was revealed by restriction fragment length polymorphism analysis and single-strand conformation polymorphism analysis. Conclusion: Exclusive homoplasmy of the 11778 mtDNA mutation in Chinese LHON patients was found in this study. Homoplasmy of the 11778 mtDNA mutation cannot account for the variation in the clinical phenotype of Chinese Leber's patients.

Original languageEnglish
Pages (from-to)196-200
Number of pages5
JournalJapanese Journal of Ophthalmology
Volume43
Issue number3
DOIs
StatePublished - 05 1999
Externally publishedYes

Keywords

  • 11778 mutation
  • Chinese Leber's hereditary optic neuropathy
  • Homoplasmy
  • Mitochondrial DNA

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