Human insulin receptors mutated at the ATP-binding site lack protein tyrosine kinase activity and fail to mediate postreceptor effects of insulin

C. K. Chou, T. J. Dull, D. S. Russell, R. Gherzi, D. Lebwohl, A. Ullrich, O. M. Rosen

Research output: Contribution to journalJournal Article peer-review

511 Scopus citations

Fingerprint

Dive into the research topics of 'Human insulin receptors mutated at the ATP-binding site lack protein tyrosine kinase activity and fail to mediate postreceptor effects of insulin'. Together they form a unique fingerprint.

Biochemistry, Genetics and Molecular Biology

Immunology and Microbiology

Pharmacology, Toxicology and Pharmaceutical Science