Improving bone microarchitecture in aging with diosgenin treatment: A study in senescence-accelerated OXYS rats

Maria A. Tikhonova, Che Hao Ting, Nataliya G. Kolosova, Chao Yu Hsu, Jian Horng Chen, Chi Wen Huang, Ging Ting Tseng, Ching Sui Hung, Pan Fu Kao, Tamara G. Amstislavskaya, Ying Jui Ho*

*Corresponding author for this work

Research output: Contribution to journalJournal Article peer-review

10 Scopus citations

Abstract

Osteoporosis is a major disease associated with aging. We have previously demonstrated that diosgenin prevents osteoporosis in both menopause and D-galactose-induced aging rats. OXYS rats reveal an accelerated senescence and are used as a suitable model of osteoporosis. The aim of the present study was to analyze microarchitecture and morphological changes in femur of OXYS rats using morphological tests and microcomputed tomography scanning, and to evaluate the effects of oral administration of diosgenin at 10 and 50 mg/kg/day on femur in OXYS rats. The result showed that, compared with age-matched Wistar rats, the femur of OXYS rats revealed lower bone length, bone weight, bone volume, frame volume, frame density, void volume, porosity, external and internal diameters, cortical bone area, BV/TV, Tb.N, and Tb.Th, but higher Tb.Sp. Eight weeks of diosgenin treatment decreased porosity and Tb.Sp, but increased BV/TV, cortical bone area, Tb.N and bone mineral density, compared with OXYS rats treated with vehicle. These data reveal that microarchitecture and morphological changes in femur of OXYS rats showed osteoporotic aging features and suggest that diosgenin may have beneficial effects on aging-induced osteoporosis.

Original languageEnglish
Pages (from-to)322-331
Number of pages10
JournalChinese Journal of Physiology
Volume58
Issue number5
DOIs
StatePublished - 2015
Externally publishedYes

Bibliographical note

Publisher Copyright:
© 2015 by The Chinese Physiological Society and Airiti Press Inc.

Keywords

  • Aging
  • Bone loss
  • Diosgenin
  • Microcomputed tomography
  • OXYS rats
  • Osteoporosis

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