Lnc-IL7R alleviates PM_2.5-mediated cellular senescence and apoptosis through EZH2 recruitment in chronic obstructive pulmonary disease

Biochemistry, Genetics and Molecular Biology

• Apoptosis 100%
• Cell Aging 100%
• Transcription Factor EZH2 100%
• Regulatory Mechanism 66%
• Oxidative Stress 66%
• Epigenetics 66%
• Cell Damage 66%
• Staining 33%
• Nested Gene 33%
• Cell Cycle 33%
• Modulation 33%
• Species 33%
• Cell Proliferation 33%
• Association 33%
• Long Noncoding RNA 33%
• Tissue Section 33%
• Chromatin Immunoprecipitation 33%
• Senescence 33%
• Particulate Matter 33%
• Colony Formation 33%

Medicine and Dentistry

• Programmed Cell Death 100%
• Cellular Senescence 100%
• Chronic Obstructive Pulmonary Disease 100%
• Transcription Factor EZH2 100%
• Cells 71%
• Patient 42%
• Lung 42%
• Regulatory Mechanism 28%
• Oxidative Stress 28%
• Cell Damage 28%
• Epigenomics 28%
• Emphysema 28%
• Epithelial Cell 14%
• Cell Proliferation 14%
• Gene 14%
• Tissues 14%
• Serum 14%
• Assay 14%
• Reactive Oxygen Species 14%
• Association 14%
• Long Term Exposure 14%
• Inpatient 14%
• Cell Cycle 14%
• Injury 14%
• Senescence 14%
• Chromatin Immunoprecipitation 14%
• Lung Injury 14%
• Colony Formation 14%
• Long Untranslated RNA 14%
• Lung Emphysema 14%
• Atmospheric Aerosols 14%

Pharmacology, Toxicology and Pharmaceutical Science

• Apoptosis 100%
• Chronic Obstructive Lung Disease 100%
• Emphysema 28%
• Cell Proliferation 14%
• Chromatin Immunoprecipitation 14%
• Reactive Oxygen Metabolite 14%
• Cell Cycle 14%
• Long Term Exposure 14%
• Injury 14%
• Lung Injury 14%
• Long Untranslated RNA 14%
• Lung Emphysema 14%
• Cellular Injury 14%